Skip Navigation

QJM 2008 101(6):423-424; doi:10.1093/qjmed/hcn068
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Bannon, M.
Right arrow Search for Related Content
PubMed
Right arrow Articles by Bannon, M.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The Author 2008. Published by Oxford University Press on behalf of the Association of Physicians. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

In this month's QJM

The first 10% of the full text of this article appears below.

This month's review paper tackles a difficult but important topic: the occurrence of gastrointestinal bleeding (GI) after percutaneous coronary intervention (PCI) or in the words of Foley et al., a deadly combination. GI bleeding is relatively common after PCI (2.3% after primary angioplasty in one study, which also reported a 10% mortality following this complication). The therapeutic dilemma arises from the fact that sudden cessation of anti-platelet therapy following PCI may result in acute stent thrombosis. So, how should the risk of bleeding post-PCI be managed? Following their review of the literature, the authors conclude that those patients considered to be at particular risk . . . [Full Text of this Article]

Michael Bannon


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?